Inflammation-Generated Extracellular Matrix Fragments Drive Lung Metastasis

نویسندگان

  • Sandrine Bekaert
  • Marianne Fillet
  • Benoit Detry
  • Muriel Pichavant
  • Raphael Marée
  • Agnes Noel
  • Natacha Rocks
  • Didier Cataldo
چکیده

Mechanisms explaining the propensity of a primary tumor to metastasize to a specific site still need to be unveiled, and clinical studies support a link between chronic inflammation and cancer dissemination to specific tissues. Using different mouse models, we demonstrate the role of inflammation-generated extracellular matrix fragments ac-PGP (N-acetyl-proline-glycine-proline) on tumor cells dissemination to lung parenchyma. In mice exposed to cigarette smoke or lipopolysaccharide, lung neutrophilic inflammation produces increased levels of MMP-9 (matrix metalloproteinase 9) that contributes to collagen breakdown and allows the release of ac-PGP tripeptides. By silencing CXCR2 gene expression in tumor cells, we show that these generated ac-PGP tripeptides exert a chemotactic activity on tumor cells in vivo by binding CXCR2.

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عنوان ژورنال:

دوره 10  شماره 

صفحات  -

تاریخ انتشار 2017